HUBUNGAN ASUPAN ENERGI DAN ASUPAN LEMAK TERHADAP PROFIL LIPID PADA PENDERITA DISLIPIDEMIA

THE CORRELATION OF ENERGY INTAKE WITH FAT INTAKE ON LIPID PROFILE IN DISLIPIDEMIC PATIENTS

Authors

  • Sugini RSUD Kendal

DOI:

https://doi.org/10.35473/jgk.v11i25.10

Keywords:

Aupan Energi, Asupan Lemak, Profil Lipid, Dislipidemia

Abstract

Background :High energy intake from saturated fat increases the risk of CHD especially in women.   This   abnormallipid   profile   is   closely   related   to   the   development   process atherosclerosis.  Epidemiological,  laboratory  and  clinical  studies  show  an  association  of increased  incidence  of  cardiovascular  disease  and  other  degenerative  diseases  with  high cholesterol levels.

Method :The research design is cross-sectional. The subjects of the study were 30 employees of  Kendal  Hospital  aged  35-50  years,  suffering  from  dyslipidemia  and  not  suffering  from other metabolic or degenerative diseases based on the examination of a specialist in internal medicine. Examination of total cholesterol, LDL cholesterol were measured by vitros system chemistry analyzer 300 in the clinical pathology  laboratory of Kendal Hospital. Food recall is   done   by   using  the  food   recall   form  through   interviews   conducted   by   trained   and experienced  nutritionists.  Data  analysis  using  Shapiro  Wilk  test  and  Spearman  correlation test. 

Results: The results of the data analysis were thatthere was a strong correlation between fat intake  and  total  cholesterol  levels  (p  =  0.01,  r  =  0.593), there  was  a  correlation  between energy  intake withtotal  cholesterol  levels  (p  =  0.016,  r  =  0.434),there  was  an  intake correlationfat with LDL levels (p = 0.011, r = 0.456) and there was no correlation between energy intake and LDL levels (p = 0.1119, r = 0.291),

Conclusion: There is a correlation between energy intake and total cholesterol (p = 0.016, r = 0.434), there is a strong correlation between fat intake and total cholesterol (p = 0.01, r = 0.593) and LDL (p = 0.011, r = 0.456).

Abstrak :

Latar Belakang : Asupan tinggi energi dari lemak jenuh meningkatkan risiko CHD khususnya pada wanita. Kelainan profil lipid ini erat hubungannya dengan proses perkembangan atherosklerosis. Penelitian epidemiologi, laboratorium dan klinik menunjukkan hubungan peningkatan kejadian penyakit kardiovaskular dan penyakit degeneratif lainnya dengan kadar kolesterol yang tinggi.

Metode : Rancangan Penelitian adalah crossecional. Subyek penelitian adalah 30 karyawati RSUD Kendal umur 35 – 50 tahun.Pemeriksaan kadar kolesterol total, kadar kolesterol LDL diukur dengan vitros system chemistry analyser 300 di laboratorium patologi klinik RSUD Kendal. Asupan gizi diukur dengan metode food recall 24 dihitung dengan program Nutrisurvey. Analisis data menggunakan uji Shapiro Wilk dan uji korelasi Spearman.

Hasil dan Pembahasan : Hasil analisa data adalah ada hubungan asupan energi dengan kadar kolesterol total (p=0,016, r=0,434), ada hubungan kuat asupan lemak dengan kadar kolesterol total (p=0,01, r=0,593), ada hubungan asupan lemak dengan kadar LDL (p=0,011, r=0,456) dan tidak ada hubungan asupan energi dengan kadar LDL (p=0,1119, r=0,291), Asupan tinggi energi mengakibatkan penimbunan lemak terutama trigliserida. Hal ini akan meningkatkan VLDL dan IDL darah yang akan berujung dengan peningkatan kolesterol total. Semakin tinggi seseorang mengkonsumsi makanan berlemak, maka timbunan kadar lemak dan trigliserida di dalam tubuh akan meningkat. Hal ini akan meningkatkan VLDL dan IDL darah.

Simpulan : Ada hubungan asupan energi dengan kadar kolesterol total (p=0,016, r=0,434), ada hubungan kuat asupan lemak dengan kadar kolesterol total (p=0,01, r=0,593) dan kadar LDL (p=0,011, r=0,456).

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Published

2019-07-04

How to Cite

Sugini (2019) “HUBUNGAN ASUPAN ENERGI DAN ASUPAN LEMAK TERHADAP PROFIL LIPID PADA PENDERITA DISLIPIDEMIA : THE CORRELATION OF ENERGY INTAKE WITH FAT INTAKE ON LIPID PROFILE IN DISLIPIDEMIC PATIENTS ”, JURNAL GIZI DAN KESEHATAN, 11(26), pp. 139–145. doi: 10.35473/jgk.v11i25.10.